# Oncology Letters

**Type:** Journals  
**Canonical URL:** https://scholariq.org/journals/oncology-letters/

## Facts

| Field | Value |
| --- | --- |
| Citations | 257,967 |
| h-index | 105 |
| Homepage | http://www.spandidos-publications.com/ol |
| Open Access | true |
| ISSN-L | 1792-1074 |
| ISSNs | 1792-1074,1792-1082 |
| OpenAlex ID | https://openalex.org/S58832373 |
| Publisher | Spandidos Publishing |
| Works | 15,881 |

## Journal papers

Showing 12 of 27.

- [Impact of prior antibiotic use on the efficacy of nivolumab for non‑small cell lung cancer](https://scholariq.org/papers/impact-of-prior-antibiotic-use-on-the-efficacy-of-nivolumab-for-non-small-cell/)
- [Primary adenoid cystic carcinoma of the lung: Clinicopathological features, treatment and results](https://scholariq.org/papers/primary-adenoid-cystic-carcinoma-of-the-lung-clinicopathological-features/)
- [AGR2 is associated with gastric cancer progression and poor survival](https://scholariq.org/papers/agr2-is-associated-with-gastric-cancer-progression-and-poor-survival/)
- [lncRNA FAM83H‑AS1 is associated with the prognosis of colorectal carcinoma and promotes cell proliferation by targeting the Notch signaling pathway](https://scholariq.org/papers/lncrna-fam83h-as1-is-associated-with-the-prognosis-of-colorectal-carcinoma-and/)
- [PRMT8 demonstrates variant-specific expression in cancer cells and correlates with patient survival in breast, ovarian and gastric cancer](https://scholariq.org/papers/prmt8-demonstrates-variant-specific-expression-in-cancer-cells-and-correlates/)
- [Diagnosis of pancreatic lesions collected by endoscopic ultrasound-guided fine-needle aspiration using next-generation sequencing](https://scholariq.org/papers/diagnosis-of-pancreatic-lesions-collected-by-endoscopic-ultrasound-guided-fine/)
- [Methyltransferase‑like 1 regulates lung adenocarcinoma A549 cell proliferation and autophagy via the AKT/mTORC1 signaling pathway](https://scholariq.org/papers/methyltransferase-like-1-regulates-lung-adenocarcinoma-a549-cell-proliferation/)
- [Whole-exome sequencing identifies variants in invasive pituitary adenomas](https://scholariq.org/papers/whole-exome-sequencing-identifies-variants-in-invasive-pituitary-adenomas/)
- [MARCKSL1 promotes the proliferation, migration and invasion of lung adenocarcinoma cells](https://scholariq.org/papers/marcksl1-promotes-the-proliferation-migration-and-invasion-of-lung/)
- [Nomogram for predicting the survival of patients with malignant melanoma: A population analysis](https://scholariq.org/papers/nomogram-for-predicting-the-survival-of-patients-with-malignant-melanoma-a/)
- [The role of MRP1 in the multidrug resistance of colorectal cancer](https://scholariq.org/papers/the-role-of-mrp1-in-the-multidrug-resistance-of-colorectal-cancer/)
- [Celastrol negatively regulates cell invasion and migration ability of human osteosarcoma via downregulation of the PI3K/Akt/NF-κB signaling pathway in vitro](https://scholariq.org/papers/celastrol-negatively-regulates-cell-invasion-and-migration-ability-of-human/)

## Journal top topics

Showing 8 of 25.

- [Cancer-related molecular mechanisms research](https://scholariq.org/topics/cancer-related-molecular-mechanisms-research/)
- [MicroRNA in disease regulation](https://scholariq.org/topics/microrna-in-disease-regulation/)
- [Circular RNAs in diseases](https://scholariq.org/topics/circular-rnas-in-diseases/)
- [RNA modifications and cancer](https://scholariq.org/topics/rna-modifications-and-cancer/)
- [Cancer Cells and Metastasis](https://scholariq.org/topics/cancer-cells-and-metastasis/)
- [Epigenetics and DNA Methylation](https://scholariq.org/topics/epigenetics-and-dna-methylation/)
- [Cancer, Hypoxia, and Metabolism](https://scholariq.org/topics/cancer-hypoxia-and-metabolism/)
- [Lung Cancer Treatments and Mutations](https://scholariq.org/topics/lung-cancer-treatments-and-mutations/)

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Source: ScholarIQ — public research metadata, principally OpenAlex. See https://scholariq.org/sources/ for provenance and https://scholariq.org/methodology/ for what these figures mean.