# David Kavanagh

**Type:** Researchers  
**Canonical URL:** https://scholariq.org/researchers/david-kavanagh/

## Facts

| Field | Value |
| --- | --- |
| Citations | 25,869 |
| Field | Complement system in diseases |
| h-index | 68 |
| i10-index | 151 |
| Last Known Institution | Newcastle University |
| OpenAlex ID | https://openalex.org/A5030805478 |
| ORCID iD | 0000-0003-4718-0072 |
| Works | 292 |

## Researcher papers

- [Biological insights from 108 schizophrenia-associated genetic loci](https://scholariq.org/papers/biological-insights-from-108-schizophrenia-associated-genetic-loci/)
- [The ExAC browser: displaying reference data information from over 60 000 exomes](https://scholariq.org/papers/the-exac-browser-displaying-reference-data-information-from-over-60-000-exomes/)
- [Genetics of HUS: the impact of MCP, CFH, and IF mutations on clinical presentation, response to treatment, and outcome](https://scholariq.org/papers/genetics-of-hus-the-impact-of-mcp-cfh-and-if-mutations-on-clinical-presentation/)
- [Atypical hemolytic uremic syndrome and C3 glomerulopathy: conclusions from a “Kidney Disease: Improving Global Outcomes” (KDIGO) Controversies Conference](https://scholariq.org/papers/atypical-hemolytic-uremic-syndrome-and-c3-glomerulopathy-conclusions-from-a/)
- [Mutations in Complement Factor I Predispose to Development of Atypical Hemolytic Uremic Syndrome](https://scholariq.org/papers/mutations-in-complement-factor-i-predispose-to-development-of-atypical-hemolytic/)
- [C-terminal truncations in human 3′-5′ DNA exonuclease TREX1 cause autosomal dominant retinal vasculopathy with cerebral leukodystrophy](https://scholariq.org/papers/c-terminal-truncations-in-human-3-5-dna-exonuclease-trex1-cause-autosomal/)
- [C3 glomerulopathy — understanding a rare complement-driven renal disease](https://scholariq.org/papers/c3-glomerulopathy-understanding-a-rare-complement-driven-renal-disease/)
- [Atypical aHUS: State of the art](https://scholariq.org/papers/atypical-ahus-state-of-the-art/)
- [The role of complement in kidney disease: conclusions from a Kidney Disease: Improving Global Outcomes (KDIGO) Controversies Conference](https://scholariq.org/papers/the-role-of-complement-in-kidney-disease-conclusions-from-a-kidney-disease/)
- [Effects of rare kidney diseases on kidney failure: a longitudinal analysis of the UK National Registry of Rare Kidney Diseases (RaDaR) cohort](https://scholariq.org/papers/effects-of-rare-kidney-diseases-on-kidney-failure-a-longitudinal-analysis-of-the/)
- [Description and Cross-Sectional Analyses of 25,880 Adults and Children in the UK National Registry of Rare Kidney Diseases Cohort](https://scholariq.org/papers/description-and-cross-sectional-analyses-of-25-880-adults-and-children-in-the-uk/)

## Researcher topics

- [Complement system in diseases](https://scholariq.org/topics/complement-system-in-diseases/)
- [Renal Diseases and Glomerulopathies](https://scholariq.org/topics/renal-diseases-and-glomerulopathies/)
- [Blood groups and transfusion](https://scholariq.org/topics/blood-groups-and-transfusion/)
- [Retinal Diseases and Treatments](https://scholariq.org/topics/retinal-diseases-and-treatments/)
- [Erythrocyte Function and Pathophysiology](https://scholariq.org/topics/erythrocyte-function-and-pathophysiology/)

## Researcher university

- [Newcastle University](https://scholariq.org/institutions/newcastle-university/)

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Source: ScholarIQ — public research metadata, principally OpenAlex. See https://scholariq.org/sources/ for provenance and https://scholariq.org/methodology/ for what these figures mean.