# Lieven Lagae

**Type:** Researchers  
**Canonical URL:** https://scholariq.org/researchers/lieven-lagae/

## Facts

| Field | Value |
| --- | --- |
| Citations | 25,881 |
| Field | Epilepsy research and treatment |
| h-index | 78 |
| i10-index | 242 |
| Last Known Institution | Allen Institute for Brain Science |
| OpenAlex ID | https://openalex.org/A5084704668 |
| ORCID iD | 0000-0002-7118-0139 |
| Works | 514 |

## Researcher papers

Showing 12 of 15.

- [Operational classification of seizure types by the International League Against Epilepsy: Position Paper of the ILAE Commission for Classification and Terminology](https://scholariq.org/papers/operational-classification-of-seizure-types-by-the-international-league-against/)
- [De Novo Mutations in the Sodium-Channel Gene SCN1A Cause Severe Myoclonic Epilepsy of Infancy](https://scholariq.org/papers/de-novo-mutations-in-the-sodium-channel-gene-scn1a-cause-severe-myoclonic/)
- [Instruction manual for the <scp>ILAE</scp> 2017 operational classification of seizure types](https://scholariq.org/papers/instruction-manual-for-the-scp-ilae-scp-2017-operational-classification-of/)
- [Mutations involved in Aicardi-Goutières syndrome implicate SAMHD1 as regulator of the innate immune response](https://scholariq.org/papers/mutations-involved-in-aicardi-goutieres-syndrome-implicate-samhd1-as-regulator/)
- [Characterization of human disease phenotypes associated with mutations in <i>TREX1</i>, <i>RNASEH2A</i>, <i>RNASEH2B</i>, <i>RNASEH2C</i>, <i>SAMHD1</i>, <i>ADAR</i>, and <i>IFIH1</i>](https://scholariq.org/papers/characterization-of-human-disease-phenotypes-associated-with-mutations-in-i/)
- [Gain-of-function mutations in IFIH1 cause a spectrum of human disease phenotypes associated with upregulated type I interferon signaling](https://scholariq.org/papers/gain-of-function-mutations-in-ifih1-cause-a-spectrum-of-human-disease-phenotypes/)
- [Genetic and phenotypic heterogeneity suggest therapeutic implications in SCN2A-related disorders](https://scholariq.org/papers/genetic-and-phenotypic-heterogeneity-suggest-therapeutic-implications-in-scn2a/)
- [Proposed consensus definitions for new‐onset refractory status epilepticus (NORSE), febrile infection‐related epilepsy syndrome (FIRES), and related conditions](https://scholariq.org/papers/proposed-consensus-definitions-for-new-onset-refractory-status-epilepticus-norse/)
- [<i>KCNQ2</i> encephalopathy: Emerging phenotype of a neonatal epileptic encephalopathy](https://scholariq.org/papers/i-kcnq2-i-encephalopathy-emerging-phenotype-of-a-neonatal-epileptic/)
- [Clinical and Molecular Phenotype of Aicardi-Goutières Syndrome](https://scholariq.org/papers/clinical-and-molecular-phenotype-of-aicardi-goutieres-syndrome/)
- [Fenfluramine hydrochloride for the treatment of seizures in Dravet syndrome: a randomised, double-blind, placebo-controlled trial](https://scholariq.org/papers/fenfluramine-hydrochloride-for-the-treatment-of-seizures-in-dravet-syndrome-a/)
- [Updated classification of epileptic seizures: Position paper of the International League Against Epilepsy](https://scholariq.org/papers/updated-classification-of-epileptic-seizures-position-paper-of-the-international/)

## Researcher topics

- [Epilepsy research and treatment](https://scholariq.org/topics/epilepsy-research-and-treatment/)
- [Pharmacological Effects and Toxicity Studies](https://scholariq.org/topics/pharmacological-effects-and-toxicity-studies/)
- [EEG and Brain-Computer Interfaces](https://scholariq.org/topics/eeg-and-brain-computer-interfaces/)
- [Neuroscience and Neuropharmacology Research](https://scholariq.org/topics/neuroscience-and-neuropharmacology-research/)
- [Neonatal and fetal brain pathology](https://scholariq.org/topics/neonatal-and-fetal-brain-pathology/)

## Researcher university

- [Allen Institute for Brain Science](https://scholariq.org/institutions/allen-institute-for-brain-science/)

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Source: ScholarIQ — public research metadata, principally OpenAlex. See https://scholariq.org/sources/ for provenance and https://scholariq.org/methodology/ for what these figures mean.