# Shizhong Zheng

**Type:** Researchers  
**Canonical URL:** https://scholariq.org/researchers/shizhong-zheng/

## Facts

| Field | Value |
| --- | --- |
| Citations | 12,667 |
| Field | Liver physiology and pathology |
| h-index | 57 |
| i10-index | 153 |
| Last Known Institution | Nanjing University of Chinese Medicine |
| OpenAlex ID | https://openalex.org/A5074514967 |
| ORCID iD | 0000-0002-7092-7843 |
| Works | 256 |

## Researcher papers

Showing 12 of 17.

- [Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)<sup>1</sup>](https://scholariq.org/papers/guidelines-for-the-use-and-interpretation-of-assays-for-monitoring-autophagy-4th/)
- [Activation of ferritinophagy is required for the RNA-binding protein ELAVL1/HuR to regulate ferroptosis in hepatic stellate cells](https://scholariq.org/papers/activation-of-ferritinophagy-is-required-for-the-rna-binding-protein-elavl1-hur/)
- [RNA-binding protein ZFP36/TTP protects against ferroptosis by regulating autophagy signaling pathway in hepatic stellate cells](https://scholariq.org/papers/rna-binding-protein-zfp36-ttp-protects-against-ferroptosis-by-regulating/)
- [Curcumin Protects the Rat Liver from CCl4-Caused Injury and Fibrogenesis by Attenuating Oxidative Stress and Suppressing Inflammation](https://scholariq.org/papers/curcumin-protects-the-rat-liver-from-ccl4-caused-injury-and-fibrogenesis-by/)
- [Isoliquiritigenin alleviates early brain injury after experimental intracerebral hemorrhage via suppressing ROS- and/or NF-κB-mediated NLRP3 inflammasome activation by promoting Nrf2 antioxidant pathway](https://scholariq.org/papers/isoliquiritigenin-alleviates-early-brain-injury-after-experimental-intracerebral/)
- [RETRACTED: Curcumin blunts epithelial-mesenchymal transition of hepatocytes to alleviate hepatic fibrosis through regulating oxidative stress and autophagy](https://scholariq.org/papers/retracted-curcumin-blunts-epithelial-mesenchymal-transition-of-hepatocytes-to/)
- [N6-methyladenosine modification regulates ferroptosis through autophagy signaling pathway in hepatic stellate cells](https://scholariq.org/papers/n6-methyladenosine-modification-regulates-ferroptosis-through-autophagy/)
- [The BRD7-P53-SLC25A28 axis regulates ferroptosis in hepatic stellate cells](https://scholariq.org/papers/the-brd7-p53-slc25a28-axis-regulates-ferroptosis-in-hepatic-stellate-cells/)
- [Canonical Wnt signaling promotes HSC glycolysis and liver fibrosis through an LDH-A/HIF-1α transcriptional complex](https://scholariq.org/papers/canonical-wnt-signaling-promotes-hsc-glycolysis-and-liver-fibrosis-through-an/)
- [Curcumol induces RIPK1/RIPK3 complex-dependent necroptosis via JNK1/2-ROS signaling in hepatic stellate cells](https://scholariq.org/papers/curcumol-induces-ripk1-ripk3-complex-dependent-necroptosis-via-jnk1-2-ros/)
- [m6A methylation is required for dihydroartemisinin to alleviate liver fibrosis by inducing ferroptosis in hepatic stellate cells](https://scholariq.org/papers/m6a-methylation-is-required-for-dihydroartemisinin-to-alleviate-liver-fibrosis/)
- [ROS-JNK1/2-dependent activation of autophagy is required for the induction of anti-inflammatory effect of dihydroartemisinin in liver fibrosis](https://scholariq.org/papers/ros-jnk1-2-dependent-activation-of-autophagy-is-required-for-the-induction-of/)

## Researcher topics

- [Liver physiology and pathology](https://scholariq.org/topics/liver-physiology-and-pathology/)
- [Liver Disease Diagnosis and Treatment](https://scholariq.org/topics/liver-disease-diagnosis-and-treatment/)
- [Traditional Chinese Medicine Analysis](https://scholariq.org/topics/traditional-chinese-medicine-analysis/)
- [Drug-Induced Hepatotoxicity and Protection](https://scholariq.org/topics/drug-induced-hepatotoxicity-and-protection/)
- [Natural product bioactivities and synthesis](https://scholariq.org/topics/natural-product-bioactivities-and-synthesis/)

## Researcher university

- [Nanjing University of Chinese Medicine](https://scholariq.org/institutions/nanjing-university-of-chinese-medicine/)

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Source: ScholarIQ — public research metadata, principally OpenAlex. See https://scholariq.org/sources/ for provenance and https://scholariq.org/methodology/ for what these figures mean.