# Yanick J. Crow

**Type:** Researchers  
**Canonical URL:** https://scholariq.org/researchers/yanick-j-crow/

## Facts

| Field | Value |
| --- | --- |
| Citations | 32,259 |
| Field | interferon and immune responses |
| h-index | 93 |
| i10-index | 242 |
| Last Known Institution | Inserm |
| OpenAlex ID | https://openalex.org/A5013758087 |
| ORCID iD | https://orcid.org/0000-0001-7211-7564 |
| Works | 424 |

## Researcher papers

- [International Union of Immunological Societies: 2017 Primary Immunodeficiency Diseases Committee Report on Inborn Errors of Immunity](https://scholariq.org/papers/international-union-of-immunological-societies-2017-primary-immunodeficiency/)
- [Mutations involved in Aicardi-Goutières syndrome implicate SAMHD1 as regulator of the innate immune response](https://scholariq.org/papers/mutations-involved-in-aicardi-goutieres-syndrome-implicate-samhd1-as-regulator/)
- [Characterization of human disease phenotypes associated with mutations in <i>TREX1</i>, <i>RNASEH2A</i>, <i>RNASEH2B</i>, <i>RNASEH2C</i>, <i>SAMHD1</i>, <i>ADAR</i>, and <i>IFIH1</i>](https://scholariq.org/papers/characterization-of-human-disease-phenotypes-associated-with-mutations-in-i/)
- [Gain-of-function mutations in IFIH1 cause a spectrum of human disease phenotypes associated with upregulated type I interferon signaling](https://scholariq.org/papers/gain-of-function-mutations-in-ifih1-cause-a-spectrum-of-human-disease-phenotypes/)
- [The 2017 IUIS Phenotypic Classification for Primary Immunodeficiencies](https://scholariq.org/papers/the-2017-iuis-phenotypic-classification-for-primary-immunodeficiencies/)
- [Preexisting autoantibodies to type I IFNs underlie critical COVID-19 pneumonia in patients with APS-1](https://scholariq.org/papers/preexisting-autoantibodies-to-type-i-ifns-underlie-critical-covid-19-pneumonia/)
- [Inflammatory profiles across the spectrum of disease reveal a distinct role for GM-CSF in severe COVID-19](https://scholariq.org/papers/inflammatory-profiles-across-the-spectrum-of-disease-reveal-a-distinct-role-for/)
- [Mutations in <i>COPA</i> lead to abnormal trafficking of STING to the Golgi and interferon signaling](https://scholariq.org/papers/mutations-in-i-copa-i-lead-to-abnormal-trafficking-of-sting-to-the-golgi-and/)
- [Protein Kinase Cδ Deficiency Causes Mendelian Systemic Lupus Erythematosus With B Cell‐Defective Apoptosis and Hyperproliferation](https://scholariq.org/papers/protein-kinase-c-deficiency-causes-mendelian-systemic-lupus-erythematosus-with-b/)

## Researcher topics

- [interferon and immune responses](https://scholariq.org/topics/interferon-and-immune-responses/)
- [Inflammasome and immune disorders](https://scholariq.org/topics/inflammasome-and-immune-disorders/)
- [Systemic Lupus Erythematosus Research](https://scholariq.org/topics/systemic-lupus-erythematosus-research/)
- [RNA regulation and disease](https://scholariq.org/topics/rna-regulation-and-disease/)
- [Immunodeficiency and Autoimmune Disorders](https://scholariq.org/topics/immunodeficiency-and-autoimmune-disorders/)

## Researcher university

- [Inserm](https://scholariq.org/institutions/inserm/)

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Source: ScholarIQ — public research metadata, principally OpenAlex. See https://scholariq.org/sources/ for provenance and https://scholariq.org/methodology/ for what these figures mean.
