# Yuka Maeda

**Type:** Researchers  
**Canonical URL:** https://scholariq.org/researchers/yuka-maeda/

## Facts

| Field | Value |
| --- | --- |
| Citations | 12,705 |
| Field | Immune Cell Function and Interaction |
| h-index | 24 |
| i10-index | 30 |
| Last Known Institution | University of Tsukuba |
| OpenAlex ID | https://openalex.org/A5024422309 |
| Works | 59 |

## Researcher papers

- [Mutational landscape determines sensitivity to PD-1 blockade in non–small cell lung cancer](https://scholariq.org/papers/mutational-landscape-determines-sensitivity-to-pd-1-blockade-in-non-small-cell/)
- [Two FOXP3+CD4+ T cell subpopulations distinctly control the prognosis of colorectal cancers](https://scholariq.org/papers/two-foxp3-cd4-t-cell-subpopulations-distinctly-control-the-prognosis-of/)
- [Lactic acid promotes PD-1 expression in regulatory T cells in highly glycolytic tumor microenvironments](https://scholariq.org/papers/lactic-acid-promotes-pd-1-expression-in-regulatory-t-cells-in-highly-glycolytic/)
- [The PD-1 expression balance between effector and regulatory T cells predicts the clinical efficacy of PD-1 blockade therapies](https://scholariq.org/papers/the-pd-1-expression-balance-between-effector-and-regulatory-t-cells-predicts-the/)
- [Anti-CCR4 mAb selectively depletes effector-type FoxP3 <sup>+</sup> CD4 <sup>+</sup> regulatory T cells, evoking antitumor immune responses in humans](https://scholariq.org/papers/anti-ccr4-mab-selectively-depletes-effector-type-foxp3-sup-sup-cd4-sup-sup/)

## Researcher topics

- [Immune Cell Function and Interaction](https://scholariq.org/topics/immune-cell-function-and-interaction/)
- [T-cell and B-cell Immunology](https://scholariq.org/topics/t-cell-and-b-cell-immunology/)
- [Immunotherapy and Immune Responses](https://scholariq.org/topics/immunotherapy-and-immune-responses/)
- [Cancer Immunotherapy and Biomarkers](https://scholariq.org/topics/cancer-immunotherapy-and-biomarkers/)
- [HER2/EGFR in Cancer Research](https://scholariq.org/topics/her2-egfr-in-cancer-research/)

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Source: ScholarIQ — public research metadata, principally OpenAlex. See https://scholariq.org/sources/ for provenance and https://scholariq.org/methodology/ for what these figures mean.