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Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model

PaperCitations, authors & open-access status

Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model is a paper indexed in ScholarIQ from OpenAlex.

How many citations does Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model have?

ScholarIQcitation count

Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model has 574 citations in the OpenAlex counts ScholarIQ stores.

When was Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model published?

ScholarIQyear

Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model has 2020 year in the ScholarIQ index.

What is the open-access status of Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model?

ScholarIQoa status

Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model has green oa status in the ScholarIQ index.

What is the DOI of Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model?

ScholarIQdoi

The DOI for Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model is on the source record.

What is the PMID of Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model?

ScholarIQpmid

The PMID for Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model is on the source record.

What is the OpenAlex record for Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model?

ScholarIQopenalex

The OpenAlex for Anti-human TREM2 induces microglia proliferation and reduces pathology in an Alzheimer’s disease model is on the source record.

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