High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism
High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism is a paper indexed in ScholarIQ from OpenAlex.
How many citations does High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism have?
High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism has 282 citations in the OpenAlex counts ScholarIQ stores.
When was High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism published?
High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism has 2018 year in the ScholarIQ index.
What is the open-access status of High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism?
High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism has closed oa status in the ScholarIQ index.
What is the DOI of High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism?
The DOI for High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism is on the source record.
What is the PMID of High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism?
The PMID for High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism is on the source record.
What is the OpenAlex record for High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism?
The OpenAlex for High salt intake causes leptin resistance and obesity in mice by stimulating endogenous fructose production and metabolism is on the source record.