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MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells

PaperCitations, authors & open-access status

MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells is a paper indexed in ScholarIQ from OpenAlex.

How many citations does MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells have?

ScholarIQcitation count

MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells has 84 citations in the OpenAlex counts ScholarIQ stores.

When was MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells published?

ScholarIQyear

MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells has 2017 year in the ScholarIQ index.

What is the open-access status of MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells?

ScholarIQoa status

MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells has closed oa status in the ScholarIQ index.

What is the DOI of MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells?

ScholarIQdoi

The DOI for MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells is on the source record.

What is the PMID of MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells?

ScholarIQpmid

The PMID for MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells is on the source record.

What is the OpenAlex record for MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells?

ScholarIQopenalex

The OpenAlex for MicroRNA-200c binding to FN1 suppresses the proliferation, migration and invasion of gastric cancer cells is on the source record.

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