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MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death

PaperCitations, authors & open-access status

MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death is a paper indexed in ScholarIQ from OpenAlex.

How many citations does MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death have?

ScholarIQcitation count

MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death has 423 citations in the OpenAlex counts ScholarIQ stores.

When was MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death published?

ScholarIQyear

MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death has 2012 year in the ScholarIQ index.

What is the open-access status of MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death?

ScholarIQoa status

MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death has bronze oa status in the ScholarIQ index.

What is the DOI of MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death?

ScholarIQdoi

The DOI for MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death is on the source record.

What is the PMID of MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death?

ScholarIQpmid

The PMID for MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death is on the source record.

What is the OpenAlex record for MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death?

ScholarIQopenalex

The OpenAlex for MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death is on the source record.

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