ScholarIQanswers from OpenAlex

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques

PaperCitations, authors & open-access status

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is a paper indexed in ScholarIQ from OpenAlex.

How many citations does Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques have?

ScholarIQcitation count

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques has 33 citations in the OpenAlex counts ScholarIQ stores.

When was Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques published?

ScholarIQyear

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques has 2015 year in the ScholarIQ index.

What is the open-access status of Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

ScholarIQoa status

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques has bronze oa status in the ScholarIQ index.

What is the DOI of Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

ScholarIQdoi

The DOI for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is on the source record.

What is the PMID of Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

ScholarIQpmid

The PMID for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is on the source record.

What is the OpenAlex record for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

ScholarIQopenalex

The OpenAlex for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is on the source record.

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