Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells
Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells is a paper indexed in ScholarIQ from OpenAlex.
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Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells has 168 citations in the OpenAlex counts ScholarIQ stores.
When was Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells published?
Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells has 2013 year in the ScholarIQ index.
What is the open-access status of Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells?
Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells has bronze oa status in the ScholarIQ index.
What is the DOI of Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells?
The DOI for Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells is on the source record.
What is the PMID of Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells?
The PMID for Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells is on the source record.
What is the OpenAlex record for Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells?
The OpenAlex for Sorafenib perpetuates cellular anticancer effector functions by modulating the crosstalk between macrophages and natural killer cells is on the source record.