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The murine homolog of<i>SALL4</i>, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with<i>Sall1</i>in anorectal, heart, brain and kidney development

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The murine homolog of<i>SALL4</i>, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with<i>Sall1</i>in anorectal, heart, brain and kidney development is a paper indexed in ScholarIQ from OpenAlex.

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The murine homolog of<i>SALL4</i>, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with<i>Sall1</i>in anorectal, heart, brain and kidney development has 266 citations in the OpenAlex counts ScholarIQ stores.

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The murine homolog of<i>SALL4</i>, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with<i>Sall1</i>in anorectal, heart, brain and kidney development has 2006 year in the ScholarIQ index.

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The murine homolog of<i>SALL4</i>, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with<i>Sall1</i>in anorectal, heart, brain and kidney development has closed oa status in the ScholarIQ index.

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The PMID for The murine homolog of<i>SALL4</i>, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with<i>Sall1</i>in anorectal, heart, brain and kidney development is on the source record.

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