ScholarIQanswers from OpenAlex & ORCID
David T. Scadden
ResearcherPublications, citations & collaboration network
David T. Scadden is a researcher indexed in ScholarIQ from OpenAlex & ORCID.
How many works does David T. Scadden have?
ScholarIQindexed works
David T. Scadden has 666 works in the ScholarIQ index. The count is the OpenAlex total, not the number of papers listed on this page.
How many citations does David T. Scadden have?
ScholarIQcitation count
David T. Scadden has 68,479 citations in the OpenAlex counts ScholarIQ stores.
What is the h-index of David T. Scadden?
ScholarIQh-index
David T. Scadden has an h-index of 124 in OpenAlex.
What is the i10-index of David T. Scadden?
ScholarIQi10-index
David T. Scadden has an i10-index of 339 in OpenAlex.
What is the ORCID of David T. Scadden?
ScholarIQorcid
The ORCID for David T. Scadden is on the source record.
What is the OpenAlex record for David T. Scadden?
ScholarIQopenalex
The OpenAlex for David T. Scadden is on the source record.
What are the most-cited papers on David T. Scadden?
ScholarIQmost cited works
BM mesenchymal stromal cell–derived exosomes facilitate multiple myeloma progression
Aldo M. Roccaro, Antonio Sacco, Patricia Maiso, Abdel Kareem Azab, Yu-Tzu Tai, Michaela R. Reagan, Feda Azab, Ludmila M. Flores, Federico Campigotto, Edie Weller, Kenneth C. Anderson, David T. Scadden, Irene M. Ghobrial
The leukemic stem cell niche: current concepts and therapeutic opportunities
Steven Lane, David T. Scadden, D. Gary Gilliland
AKT/FOXO Signaling Enforces Reversible Differentiation Blockade in Myeloid Leukemias
Stephen M. Sykes, Steven Lane, Lars Bullinger, Demetrios Kalaitzidis, Rushdia Z. Yusuf, Borja Sáez, Francesca Ferraro, François Mercier, Harshabad Singh, Kristina Brumme, Sanket S. Acharya, Claudia Scholl, Zuzana Tóthová, Eyal C. Attar, Stefan Fröhling, Ronald A. DePinho, D. Gary Gilliland, Scott A. Armstrong, David T. Scadden
Preclinical modeling highlights the therapeutic potential of hematopoietic stem cell gene editing for correction of SCID-X1
Giulia Schiroli, Samuele Ferrari, Anthony Conway, Aurélien Jacob, Valentina Capo, Luisa Albano, Tiziana Plati, Maria Carmina Castiello, Francesca Sanvito, Andrew R. Gennery, Chiara Bovolenta, Rahul Palchaudhuri, David T. Scadden, Michael C. Holmes, Anna Villa, Giovanni Sitia, Angelo Lombardo, Pietro Genovese, Luigi Naldini
Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney disease
Eunsil Hahm, Changli Wei, Isabel Cuesta Fernández, Jing Li, Nicholas J. Tardi, Melissa Tracy, Shikha Wadhwani, Yanxia Cao, Vasil Peev, Andrew Zloza, Jevgenijs Lusciks, Salim S. Hayek, Christopher L. O’Connor, Markus Bitzer, Vineet Gupta, Sanja Sever, David B. Sykes, David T. Scadden, Jochen Reiser