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Hartmut Halfter
ResearcherPublications, citations & collaboration network
Hartmut Halfter is a researcher indexed in ScholarIQ from OpenAlex & ORCID. ScholarIQ records 50 works, 2,387 citations, an h-index of 25 and an i10-index of 35.
50
Works
2,387
Citations
25
h-index
35
i10-index
IDs:OpenAlex
How has Hartmut Halfter's publication output changed over time?
ScholarIQpublication output · 1989–2014
Output grew0% over the shown period — from 2 works in 1989 to 2 in 2014.
2
1
1
1
1
1
1
2
19891993200020032004200520102014
What are the most-cited papers on Hartmut Halfter?
ScholarIQmost cited works
Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathways
Masao Mizuki, R. Fenski, Hartmut Halfter, Itaru Matsumura, Rainer Schmidt, Carsten Müller‐Tidow, W Grüning, Karsten Kratz‐Albers, S Serve, Claudia Steur, Thomas Büchner, Joachim Kienast, Yuzuru Kanakura, Wolfgang E. Berdel, Hubert Serve
Blood. 2000584 Citations
Mutations in SEPT9 cause hereditary neuralgic amyotrophy
Gregor Kuhlenbäumer, Mark Hannibal, Eva Nelis, Anja Schirmacher, Nathalie Verpoorten, Jan Meuleman, Giles D. Watts, Els De Vriendt, Peter Young, Florian Stögbauer, Hartmut Halfter, Joy Irobi, D. Goossens, Jurgen Del‐Favero, Benjamin G Betz, Hyun Hor, G. Kurlemann, Thomas D. Bird, Eila Airaksinen, Tarja Mononen, Adolfo Pou Serradell, J M Prats, Christine Van Broeckhoven, Peter De Jonghe, Vincent Timmerman, E B Ringelstein, Phillip F. Chance
S137905309. 2005266 Citations
Tyrosine 785 is a major determinant of Trk‐‐substrate interaction.
Axel Obermeier, Hartmut Halfter, Karl‐Heinz Wiesmüller, Gwanghyun Jung, Joseph Schlessinger, A. Ullrich
S127916151. 1993186 CitationsOPEN ACCESS
Delineating an oncostatin M-activated STAT3 signaling pathway that coordinates the expression of genes involved in cell cycle regulation and extracellular matrix deposition of MCF-7 cells
Fang Zhang, Cong Li, Hartmut Halfter, Jingwen Liu
S128439998. 2003136 Citations
High-Throughput Analysis of Genome-Wide Receptor Tyrosine Kinase Expression in Human Cancers Identifies Potential Novel Drug Targets
Carsten Müller‐Tidow, Joachim Schwäble, Björn Steffen, Nicola Tidow, B. Brandt, Kerstin Becker, Eric Schulze‐Bahr, Hartmut Halfter, U Vogt, Ralf Metzger, Paul M. Schneider, Thomas Büchner, Christian Brandts, Wolfgang E. Berdel, Hubert Serve
Clinical Cancer Research. 2004119 Citations
Related on ScholarIQ
University Hospital Münster
Institution
Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathways
Paper
Mutations in SEPT9 cause hereditary neuralgic amyotrophy
Paper
Tyrosine 785 is a major determinant of Trk‐‐substrate interaction.
Paper
Delineating an oncostatin M-activated STAT3 signaling pathway that coordinates the expression of genes involved in cell cycle regulation and extracellular matrix deposition of MCF-7 cells
Paper
High-Throughput Analysis of Genome-Wide Receptor Tyrosine Kinase Expression in Human Cancers Identifies Potential Novel Drug Targets
Paper