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About the database ScholarIQanswers from OpenAlex & ORCID
How has Robert M. Graham's publication output changed over time?
ScholarIQpublication output · 1996–2019
Output grew0% over the shown period — from 1 works in 1996 to 1 in 2019.
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1996199920022003200720092010201420152019
What are the most-cited papers on Robert M. Graham?
ScholarIQmost cited works
Transglutaminases: crosslinking enzymes with pleiotropic functions
L. Lóránd, Robert M. Graham
S196724125. 20031,430 Citations
G <sub>h</sub> : a GTP-Binding Protein with Transglutaminase Activity and Receptor Signaling Function
Hideaki Nakaoka, Dianne M. Perez, Kwang Jin Baek, Tanya Das, Ahsan Husain, Kunio S. Misono, Mie-Jae Im, Robert M. Graham
Science. 1994551 Citations
α<sub>1</sub>-Adrenergic Receptor Subtypes
Robert M. Graham, Dianne M. Perez, John Hwa, Michael T. Piascik
S10652568. 1996522 Citations
Skeletal muscle hypertrophy is mediated by a Ca2+-dependent calcineurin signalling pathway
Christopher Semsarian, Mingjie Wu, Yue‐Kun Ju, Tadeusz Marciniec, Thomas Yeoh, David G. Allen, Richard P. Harvey, Robert M. Graham
Nature. 1999454 Citations
Adult heart transplantation with distant procurement and ex-vivo preservation of donor hearts after circulatory death: a case series
K. Dhital, Arjun Iyer, M. Connellan, H. Chew, Ling Gao, Aoife Doyle, Mark Hicks, G. Kumarasinghe, C. Soto, A. Dinale, Bruce Cartwright, Priya Nair, Emily Granger, P. Jansz, Andrew Jabbour, Eugene Kotlyar, Anne Keogh, Chris Hayward, Robert M. Graham, Phillip Spratt, Peter S. Macdonald
The Lancet. 2015448 Citations
Related on ScholarIQ
Victor Chang Cardiac Research Institute
Institution
Transglutaminases: crosslinking enzymes with pleiotropic functions
Paper
G <sub>h</sub> : a GTP-Binding Protein with Transglutaminase Activity and Receptor Signaling Function
Paper
α<sub>1</sub>-Adrenergic Receptor Subtypes
Paper
Skeletal muscle hypertrophy is mediated by a Ca2+-dependent calcineurin signalling pathway
Paper
Adult heart transplantation with distant procurement and ex-vivo preservation of donor hearts after circulatory death: a case series
Paper