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About the database ScholarIQanswers from OpenAlex & ORCID
How has Tao Yang's publication output changed over time?
ScholarIQpublication output · 2015–2022
Output grew0% over the shown period — from 2 works in 2015 to 2 in 2022.
2
1
1
1
2
1
1
2
20152016201720182019202020212022
What are the most-cited papers on Tao Yang?
ScholarIQmost cited works
Eye-Wearable Technology for Machine Maintenance
Xianjun Sam Zheng, Cédric Foucault, Patrik Matos da Silva, Siddharth Dasari, Tao Yang, Stuart Goose
201590 Citations
lncRNA PVT1 and its splicing variant function as competing endogenous RNA to regulate clear cell renal cell carcinoma progression
Tao Yang, Hui Zhou, Peijun Liu, Libin Yan, Weimin Yao, Ke Chen, Jin Zeng, Heng Li, Junhui Hu, Hua Xu, Zhangqun Ye
Oncotarget. 201768 CitationsOPEN ACCESS
Huachansu suppresses human bladder cancer cell growth through the Fas/Fasl and TNF- alpha/TNFR1 pathway in vitro and in vivo
Tao Yang, Runlin Shi, Lei Chang, Kun Tang, Ke Chen, Yu Gan, Yuanfeng Tian, Yonglian Guo, Wei He, Xiaodong Song, Hua Xu, Zhangqun Ye
Journal of Experimental & Clinical Cancer Research. 201566 CitationsOPEN ACCESS
miR-122 Inhibits Hepatocarcinoma Cell Progression by Targeting LMNB2
Xiaona Li, Hong Yang, Tao Yang
S146643948. 201945 CitationsOPEN ACCESS
<i>TACC3</i> overexpression in cholangiocarcinoma correlates with poor prognosis and is a potential anti-cancer molecular drug target for HDAC inhibitors
Junchuang He, Wei Yao, Jianming Wang, Peter Schemmer, Yan Yang, Yan Liu, Yawei Qian, Weipeng Qi, Jian Zhang, Qi Shen, Tao Yang
Oncotarget. 201644 CitationsOPEN ACCESS
Related on ScholarIQ
Qingdao University
Institution
Eye-Wearable Technology for Machine Maintenance
Paper
lncRNA PVT1 and its splicing variant function as competing endogenous RNA to regulate clear cell renal cell carcinoma progression
Paper
Huachansu suppresses human bladder cancer cell growth through the Fas/Fasl and TNF- alpha/TNFR1 pathway in vitro and in vivo
Paper
miR-122 Inhibits Hepatocarcinoma Cell Progression by Targeting LMNB2
Paper
<i>TACC3</i> overexpression in cholangiocarcinoma correlates with poor prognosis and is a potential anti-cancer molecular drug target for HDAC inhibitors
Paper