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How has Thomas Weimbs's publication output changed over time?
ScholarIQpublication output · 1992–2019
Output grew0% over the shown period — from 1 works in 1992 to 1 in 2019.
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1992199619971998200320042006201020182019
What are the most-cited papers on Thomas Weimbs?
ScholarIQmost cited works
The mTOR pathway is regulated by polycystin-1, and its inhibition reverses renal cystogenesis in polycystic kidney disease
Jonathan M. Shillingford, Noel Murcia, Claire H. Larson, Seng Hui Low, Ryan C. Hedgepeth, Nicole Brown, Chris A. Flask, Andrew C. Novick, David A. Goldfarb, Albrecht Kramer-Zucker, Gerd Walz, Klaus Piontek, Gregory G. Germino, Thomas Weimbs
Proceedings of the National Academy of Sciences. 2006800 CitationsOPEN ACCESS
Polycystin-1, STAT6, and P100 Function in a Pathway that Transduces Ciliary Mechanosensation and Is Activated in Polycystic Kidney Disease
Seng Hui Low, Shivakumar Vasanth, Claire H. Larson, Sambuddho Mukherjee, Nikunj Sharma, Michael Kinter, Michelle E. Kane, Tomoko Obara, Thomas Weimbs
S83804233. 2006366 CitationsOPEN ACCESS
A conserved domain is present in different families of vesicular fusion proteins: A new superfamily
Thomas Weimbs, Seng Hui Low, Steven J. Chapin, Keith E. Mostov, Philipp Bücher, Kay Hofmann
Proceedings of the National Academy of Sciences. 1997312 CitationsOPEN ACCESS
Rapamycin Ameliorates PKD Resulting from Conditional Inactivation of Pkd1
Jonathan M. Shillingford, Klaus Piontek, Gregory G. Germino, Thomas Weimbs
Journal of the American Society of Nephrology. 2010260 CitationsOPEN ACCESS
Differential localization of syntaxin isoforms in polarized Madin-Darby canine kidney cells.
Sarah Löw, Steven J. Chapin, Thomas Weimbs, László G. Kömüves, Mark K. Bennett, Keith E. Mostov
S188085164. 1996234 CitationsOPEN ACCESS
Related on ScholarIQ
Santa Barbara City College
Institution
The mTOR pathway is regulated by polycystin-1, and its inhibition reverses renal cystogenesis in polycystic kidney disease
Paper
Polycystin-1, STAT6, and P100 Function in a Pathway that Transduces Ciliary Mechanosensation and Is Activated in Polycystic Kidney Disease
Paper
A conserved domain is present in different families of vesicular fusion proteins: A new superfamily
Paper
Rapamycin Ameliorates PKD Resulting from Conditional Inactivation of Pkd1
Paper
Differential localization of syntaxin isoforms in polarized Madin-Darby canine kidney cells.
Paper