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How has Yu‐Chi Chang's publication output changed over time?
ScholarIQpublication output · 2005–2022
Output grew0% over the shown period — from 1 works in 2005 to 1 in 2022.
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20052008201620172018201920202022
What are the most-cited papers on Yu‐Chi Chang?
ScholarIQmost cited works
Effects of air pollution on the nervous system and its possible role in neurodevelopmental and neurodegenerative disorders
Lucio G. Costa, Toby B. Cole, Khoi Dao, Yu‐Chi Chang, Jacki Coburn, Jacqueline M. Garrick
Pharmacology & Therapeutics. 2020485 CitationsOPEN ACCESS
Developmental impact of air pollution on brain function
Lucio G. Costa, Toby B. Cole, Khoi Dao, Yu‐Chi Chang, Jacqueline M. Garrick
Neurochemistry International. 2019159 CitationsOPEN ACCESS
Sex and genetic differences in the effects of acute diesel exhaust exposure on inflammation and oxidative stress in mouse brain
Toby B. Cole, Jacki Coburn, Khoi Dao, Pam Roqué, Yu‐Chi Chang, Vrinda Kalia, Tomás R. Guilarte, Jennifer L. Dziedzic, Lucio G. Costa
Toxicology. 2016129 Citations
Developmental Neurotoxicity of Traffic-Related Air Pollution: Focus on Autism
Lucio G. Costa, Yu‐Chi Chang, Toby B. Cole
Current Environmental Health Reports. 201791 CitationsOPEN ACCESS
Prenatal and early life diesel exhaust exposure disrupts cortical lamina organization: Evidence for a reelin-related pathogenic pathway induced by interleukin-6
Yu‐Chi Chang, Ray A. M. Daza, Robert F. Hevner, Lucio G. Costa, Toby B. Cole
S102243518. 201945 CitationsOPEN ACCESS
Related on ScholarIQ
National Yang Ming Chiao Tung University
Institution
Effects of air pollution on the nervous system and its possible role in neurodevelopmental and neurodegenerative disorders
Paper
Developmental impact of air pollution on brain function
Paper
Sex and genetic differences in the effects of acute diesel exhaust exposure on inflammation and oxidative stress in mouse brain
Paper
Developmental Neurotoxicity of Traffic-Related Air Pollution: Focus on Autism
Paper
Prenatal and early life diesel exhaust exposure disrupts cortical lamina organization: Evidence for a reelin-related pathogenic pathway induced by interleukin-6
Paper