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Receptor Mechanisms and Signaling

TopicLeading institutions, researchers & key papers

Receptor Mechanisms and Signaling is a topic indexed in ScholarIQ from OpenAlex.

What is known about Receptor Mechanisms and Signaling?

ScholarIQrecord summary

This cluster of papers explores the structure, function, and signaling mechanisms of G protein-coupled receptors (GPCRs), with a focus on crystal structures, signal transduction, arrestin-mediated pathways, pharmacology, drug discovery, receptor activation, biased signaling, and the molecular mechanisms involved. The research covers a wide range of topics related to GPCRs and their implications in various biological processes.

How many works does Receptor Mechanisms and Signaling have?

ScholarIQindexed works

Receptor Mechanisms and Signaling has 107,465 works in the ScholarIQ index. The count is the OpenAlex total, not the number of papers listed on this page.

How many citations does Receptor Mechanisms and Signaling have?

ScholarIQcitation count

Receptor Mechanisms and Signaling has 2,883,182 citations in the OpenAlex counts ScholarIQ stores.

What is the OpenAlex record for Receptor Mechanisms and Signaling?

ScholarIQopenalex

The OpenAlex for Receptor Mechanisms and Signaling is on the source record.

Who are the most-cited researchers behind Receptor Mechanisms and Signaling?

ScholarIQtop researchers by citations
1
Eric S. Lander · Genomics and Chromatin Dynamics
891 works519,894 citations
2
Peer Bork · Genomics and Phylogenetic Studies
950 works390,812 citations
3
Matthias Mann · Advanced Proteomics Techniques and Applications
1,456 works305,659 citations
4
Robert A. Weinberg · Cancer Cells and Metastasis
668 works298,760 citations
5
Richard A. Gibbs · Genomics and Rare Diseases
1,328 works289,961 citations

Where is Receptor Mechanisms and Signaling research published, and who funds it?

ScholarIQvenues & funding sources

TOP JOURNALS

No journal data available yet.

TOP FUNDERS

National Science Foundation—
NIH—
Wellcome Trust—
European Research Council—
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