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Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation

PaperCitations, authors & open-access status

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is a paper indexed in ScholarIQ from OpenAlex.

How many citations does Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation have?

ScholarIQcitation count

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation has 28 citations in the OpenAlex counts ScholarIQ stores.

When was Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation published?

ScholarIQyear

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation has 2023 year in the ScholarIQ index.

What is the open-access status of Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

ScholarIQoa status

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation has hybrid oa status in the ScholarIQ index.

What is the DOI of Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

ScholarIQdoi

The DOI for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is on the source record.

What is the PMID of Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

ScholarIQpmid

The PMID for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is on the source record.

What is the OpenAlex record for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

ScholarIQopenalex

The OpenAlex for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is on the source record.

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